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In order to maintain intracellular glutamine-derived glutamate levels, NRF2-addicted cells exploit glutamine transporter, SLC1A5, to uptake more extracellular glutamine that is then converted to glutamate by glutaminase (Romero et al

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Additionally, in concanavalin A- (Con A)-induced AIH, pretreatment with Fer-1 reduced the severity of Con A-induced liver lesions and the occurrence of ferroptosis events, including the elevation of hepatic GSH, GPX4, and system Xc-expressions and downregulation of ferrous iron levels in liver tissues (Zeng et al., 2020)

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